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In this regard, PK activity enhancement can be thought to mitigate the instability of the HS RBC membrane by replenishing ATP stores that are then used in the ion-pump activities (24), acceleration of glutathione (GSH) synthesis, and a feed-forward loop for glycolysis itself (7, 25, 26)

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In silico therapies at early and late time points after APAP insult indicated that prevention of necrotic damage is most beneficial for recovery, whereas interference with regulation of senescence promotes regeneration in a less pronounced way